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Discussion Summary

Paper(s): Baker, D., Wijshake, T., Tchkonia, T. et al. Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders. Nature 479, 232–236 (2011). https://doi.org/10.1038/nature10600. This was the paper that launched Unity Biotechnology. Later shown in WT mice Baker, D., Childs, B., Durik, M. et al. Naturally occurring p16Ink4a-positive cells shorten healthy lifespan. Nature 530, 184–189 (2016). https://doi.org/10.1038/nature16932.

Presented by Tara Mei on 3/27/2022

Senecent Cells and Aging

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What is a senescent cell?

Image: NIA

SASPS

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Image: Roe Ethridge

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The GOOD

Tumour suppression

Embryonic development

Wound healing

The BAD

Tumour growth

Seem to be present in lots of aging diseases

?

?

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QUESTION

What happens if you remove senescent cells?

BIGGER QUESTION

Are senescent cells causal in aging diseases?

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STEP 1: Made a transgene that removes SCs in mice upon drug treatment

STEP 2: Cleared SCs with drug

STEP 3: Checked for effects of clearing SCs on aging

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So what happened?

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Delayed lordokyphosis

Delayed cataracts

Larger muscle fibres

Larger fat deposits

Better exercise

So what happened?

…but no lifespan increase

Side effects: None

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Delayed lordokyphosis

Delayed cataracts

Larger muscle fibres

Larger fat deposits

Better exercise

What happens if you remove SCs later in life?

Not mentioned

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QUESTION

What happens if you remove senescent cells?

BIGGER QUESTION

Are senescent cells causal in aging diseases?

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So what happened?

Lifespan increase

Delayed cataracts

Larger fat deposits

Reduced glomerulosclerosis

Increased cardiac stress tolerance

Side effects: Slower wound closure

New!

New!

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n=14

n=9

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Still unclear:

Do both chronic and acute senescense matter in aging disease?

Why do senescent cells accumulate with age?

What might side effects of clearing SCs be?

Are all cell types equally responsive to senolytics?

How do we characterise senescence/how many markers are enough?

senescence-associated β-galactosidase

enlarged cytoplasm

lack of DNA replication

expression of kinase inhibitors p21 and p16

nuclear senescence-associated heterochromatin foci

SASPS