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Endocrine Revision:

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A 28-years-old woman with a history of type 1 diabetes was brought in to the emergency department due to palpitations, diaphoresis and tremors. On examination, her vital signs show a heart rate of 120 bpm and respiratory rate of 24 per min. It is noted that she has tremors of her hands, unusually bulging eyeballs and a diffuse swelling in her neck and shins. Her blood tests show:

TSH 0.1 mU/l (low)

Free T4 32.5 pmol/l (high)

Free T3 12.5 pmol/l (high)

What is the most likely underlying pathophysiology in this patient?

  1. Hashimoto’s Thyroiditis
  2. Toxic Multinodular Goitre
  3. Grave’s Disease
  4. Subacute Thyroiditis

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A 28-years-old woman with a history of type 1 diabetes was brought in to the emergency department due to palpitations, diaphoresis and tremors. On examination, her vital signs show a heart rate of 120 bpm and respiratory rate of 24 per min. It is noted that she has tremors of her hands, unusually bulging eyeballs (exophthalmos) and a diffuse swelling (pretibial myxoedema) in her neck and shins. Her blood tests show:

TSH 0.1 mU/l (low)

Free T4 32.5 pmol/l (high)

Free T3 12.5 pmol/l (high)

What is the most likely underlying pathophysiology in this patient?

  • Hashimoto’s Thyroiditis
  • Toxic Multinodular Goitre
  • Grave’s Disease
  • Subacute Thyroiditis

What are the appropriate treatment options?

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What are the appropriate first-line treatment options for Graves Disease?

Control symptoms with propranolol; antithyroid drugs like carbimazole, propylthiouracil

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A 42-year-old woman presents with a 2-week history of fever and malaise. On examination, the patient reports a dull, aching pain in the front of her neck that is painful on palpation. She has felt increasingly fatigued and noted mild palpitations and heat intolerance. She denies significant weight changes but admits to occasional sweating and irritability. Two weeks ago, she had a sore throat and mild fever, which resolved after a few days.�

TSH 0.2 mU/l (low)

Free T4 33.4 pmol/l (high)

Free T3 12.7 pmol/l (high)

�What is the most likely underlying pathophysiology in this patient?

  1. Subacute thyroiditis (De Quervain’s thyroiditis)
  2. Graves Disease
  3. Thyroid Storm
  4. Hashimoto’s Thyroiditis

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A 42-year-old woman presents with a 2-week history of fever and malaise. On examination, the patient reports a dull, aching pain in the front of her neck that is painful on palpation. She has felt increasingly fatigued and noted mild palpitations and heat intolerance. She denies significant weight changes but admits to occasional sweating and irritability. Two weeks ago, she had a sore throat and mild fever, which resolved after a few days.�

TSH 0.2 mU/l (low)

Free T4 33.4 pmol/l (high)

Free T3 12.7 pmol/l (high)

�What is the most likely underlying pathophysiology in this patient?

  • Subacute thyroiditis (De Quervain’s thyroiditis)
  • Graves Disease
  • Thyroid Storm
  • Hashimoto’s Thyroiditis

What are the appropriate treatment options?

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What are the appropriate treatment options for Subacute Thyroiditis (De Quervain’s Thyroiditis)?

Early Stage: Control symptoms with propranolol; pain control (NSAIDs/Corticosteroids)

The hyperthyroid phase is due to the release of preformed thyroid hormones from damaged thyroid follicles; it is a temporary phase that is followed by a longer hypothyroid phase

Later stage: Monitor thyroid function, possible levothyroxine replacement

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A 35-year-old woman presents with a 6-month history of fatigue, weight gain, and cold intolerance. She reports feeling unusually tired despite adequate sleep, and she has gained approximately 5 kg over the past few months despite no significant changes in her diet. She mentions that she has noticed her skin becoming dry and hair thinning. Upon examination, the patient has pale skin, bradycardia (52 bpm), and a symmetrical, smooth non-tender goiter in her neck. She recently immigrated to Scotland from a mountainous region in South Asia. Her blood tests show:

TSH (high)

Free T4 (low)

Anti-TPO Negative

What is the most likely underlying pathophysiology in this patient?

  • Hashimoto’s Thyroiditis
  • Thyroid Storm
  • Grave’s Disease
  • Iodine Deficiency

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A 35-year-old woman presents with a 6-month history of fatigue, weight gain, and cold intolerance. She reports feeling unusually tired despite adequate sleep, and she has gained approximately 5 kg over the past few months despite no significant changes in her diet. She mentions that she has noticed her skin becoming dry and hair thinning. Upon examination, the patient has pale skin, bradycardia (52 bpm), and a symmetrical, smooth non-tender goiter in her neck. She recently immigrated to Scotland from a mountainous region in South Asia. Her blood tests show:

TSH 0.1 mU/l (low)

Free T4 32.5 pmol/l (high)

Anti-TPO Negative

What is the most likely underlying pathophysiology in this patient?

  • Hashimoto’s Thyroiditis
  • Thyroid Storm
  • Grave’s Disease
  • Iodine Deficiency

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Treatment

Hyperthyroid:

Propranolol (dampens sympathetic nervous system), Propylthiouracil/carbimazole (blocks synthesis of thyroid hormones), radioiodine treatment(destroys overactive thyroid tissue), and surgery (remove overactive thyroid tissue)

Hypoparathyroidism Treatment:

Replace with levothyroxine (exogenous thyroid hormones substitute)

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Calcium regulation

The two hormones which primarily control calcium metabolism are:

  • parathyroid hormone (PTH)
  • 1,25-dihydroxycholecalciferol (calcitriol, the active form of vitamin D)

What are the actions of PTH?

What are the actions of calcitriol?

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Calcium regulation

The two hormones which primarily control calcium metabolism are:

  • parathyroid hormone (PTH)
  • 1,25-dihydroxycholecalciferol (calcitriol, the active form of vitamin D)

What are the actions of PTH?

  • increases plasma calcium, decreases plasma phosphate
  • increases renal tubular reabsorption of calcium
  • increases osteoclastic activity*
  • increases renal conversion of 25-hydroxycholecalciferol to 1,25-dihydroxycholecalciferol
  • decreases renal phosphate reabsorption

What are the actions of calcitriol?

  • increases plasma calcium, increases plasma phosphate
  • Enhances calcium absorption in the small intestine by increasing the expression of calcium-binding proteins.
  • Promotes phosphate absorption in the gut, essential for bone mineralization.

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A 67 year old patient is recovering from a thyroidectomy on the ward. The nurse comes to get you as the patient starts to complain of tingling and numbness in the face, toes, and fingers. What is the most likely underlying cause?

  1. Hyperparathyroidism
  2. Hypoparathyroidism
  3. Thyroid storm
  4. Carpal Tunnel Syndrome

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A 67 year old patient is recovering from a thyroidectomy on the ward. The nurse comes to get you as the patient starts to complain of tingling and numbness in the face, toes, and fingers. What is the most likely underlying cause?

  • Hyperparathyroidism
  • Hypoparathyroidism
  • Thyroid storm
  • Carpal Tunnel Syndrome

Hypocalcemia, as a result of hypoparathyroidism from damaged parathyroid glands during thyroidectomy

What is the treatment?

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A 67 year old patient is recovering from a thyroidectomy on the ward. The nurse comes to get you as the patient starts to complain of tingling and numbness in the face, toes, and fingers. What is the most likely underlying cause?

  • Hyperparathyroidism
  • Hypoparathyroidism
  • Thyroid storm
  • Carpal Tunnel Syndrome

Hypocalcemia, as a result of hypoparathyroidism from damaged parathyroid glands during thyroidectomy

What is the treatment?

Calcium supplementation, vitamin D might be added

Magnesium deficiency must be corrected before calcium supplementation because magnesium is essential for the proper secretion and function of PTH, and for the activation of vitamin D to enhance calcium absorption.

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A 60-year-old postmenopausal woman presents with fatigue, joint pain, and increased anxiety over the past few months. She reports recurrent kidney stones in the last 2 years. Her memory is worsening, and she feels more irritable. She has mild swelling in her knees. The patient has no history of chronic kidney disease or vitamin D deficiency. What lab value is most likely to be decreased?

  1. Calcium
  2. Parathyroid Hormone
  3. Vitamin D
  4. Phosphate

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A 60-year-old postmenopausal woman presents with fatigue, joint pain, and increased anxiety over the past few months. She reports recurrent kidney stones in the last 2 years. Her memory is worsening, and she feels more irritable. She has mild swelling in her knees. The patient has no history of chronic kidney disease or vitamin D deficiency. What lab value is most likely to be decreased?

  • Calcium
  • Parathyroid Hormone
  • Vitamin D
  • Phosphate

The patient most likely has primary hyperparathyroidism

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What do these arterial blood gas values indicate?

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What do these arterial blood gas values indicate?

Acidotic

Not due to excess

CO2

Is due to insufficient HCO3

Possibly raised anion gap?

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An 10-year-old boy with no past medical history presents to the emergency department with diarrhoea and vomiting. He complains of abdominal pain. He had a blood glucose finger-prick test which read Glucose = 24. An arterial blood gas was performed (as listed on the previous slide). The anion gap is confirmed to be elevated. What is his most likely diagnosis?

  1. Diabetic Ketoacidosis
  2. Acute Gastroenteritis with dehydration
  3. Hypoglycemia due to insulinoma
  4. Hyperosmolar hyperglycemic state

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An 10-year-old boy with no past medical history presents to the emergency department with diarrhoea and vomiting. He complains of abdominal pain. He had a blood glucose finger-prick test which read Glucose = 24. An arterial blood gas was performed (as listed on the previous slide). The anion gap is confirmed to be elevated. What is his most likely diagnosis?

  • Diabetic Ketoacidosis
  • Acute Gastroenteritis with dehydration
  • Hypoglycemia due to insulinoma
  • Hyperosmolar hyperglycemic state

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Diabetes

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DKA - Diabetic ketoacidosis

  • Uncontrolled LIPOLYSIS not proteolysis
    • excess fatty acids get converted to ketone bodies
  • Causes
    • infection, missed doses, MI
  • Features
  • “DKA is Deadly”
    • Delirium/psychosis
    • Kussmaul respirations
    • Abdominal pain/nausea/vomiting
    • Dehydration
    • Fruity breath
  • Bloods: Hyperglycemia, depleted intracellular potassium
  • Main management: Insulin, correct electrolyte disturbances, FLUIDS

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A 70-year-old woman presents to her GP complaining of weight gain over the past few months. Her friends have been commenting on her face appears rounder and developed some stretch marks on her abdomen. On examination, she is noted to have a heart rate of 99 beats per minute, a respiratory rate of 17 breaths per minute and blood pressure of 162/74 mmHg. She has a waist circumference of 41 inches and a body mass index of 31 kg/m2. Which of the following correctly describes one of the effects of the main hormone implicated in this patient's condition?

  1. Increases glucose uptake in peripheral tissues
  2. Increases lipolysis and causes central obesity
  3. Promotes protein synthesis in skeletal muscles
  4. Suppresses bone resorption and increases bone density

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A 70-year-old woman presents to her GP complaining of weight gain over the past few months. Her friends have been commenting on her face appears rounder and developed some stretch marks on her abdomen. On examination, she is noted to have a heart rate of 99 beats per minute, a respiratory rate of 17 breaths per minute and blood pressure of 162/74 mmHg. She has a waist circumference of 41 inches and a body mass index of 31 kg/m2. Which of the following correctly describes one of the effects of the main hormone implicated in this patient's condition?

  • Increases glucose uptake in peripheral tissues
  • Increases lipolysis and causes central obesity
  • Promotes protein synthesis in skeletal muscles
  • Suppresses bone resorption and increases bone density

The most likely diagnosis is Cushing’s disease which is characterised by high cortisol

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Cushings

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Endocrine HTN (5-10%)

⇡ ALDOSTERONE

⇡ CORTISOL

⇡ CATECHOLAMINES

Too little

🡪 Hypotension

Adrenaline, Noradrenaline

Hyperaldosteronism

Cushing’s

Phaeochromocytoma

SALT

SUGAR

SEX

G

F

R

Mineralocorticoids

Glucocorticoids

Androgens

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Addison’s Disease

Primary Adrenal Insufficiency

⇣ Aldosterone 🡪 Hyponatraemia, Hypotension (postural)

⇣ Cortisol 🡪 Hypoglycaemia, ⇡ ACTH

- Skin Pigmentation

Autoimmune (Hx / FH)

Early am Cortisol (Low / not v useful)

Short SynACTHen (< Double baseline)

ACTH (HIGH)

Adrenal cortex / 21-hydroxylase Abs (80%)

HYDROCORTISONE

Fludrocortisone

Glucocorticoid

Mineralocorticoid

Decreased Libido

🡪 Androgen replacement (DHEA)

*SICK DAY RULES

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A 49-year-old woman attends an appointment with her GP, she complains of night sweats, hot flashes and vaginal dryness which causes sexual intercourse to be painful. What lab test would be used to confirm the most likely diagnosis?

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A 49-year-old woman attends an appointment with her GP, she complains of night sweats, hot flashes and vaginal dryness which causes sexual intercourse to be painful. What would lab test would be used to confirm the most likely diagnosis?

Follicle-Stimulating Hormone

FSH increases due to the loss of negative feedback on FSH from lack of estrogen. It is usually diagnostic for menopause.

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Menstrual cycle

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Menopause

Main symptoms:

  • Periods
    • change in length
    • irregular
  • Vasomotor
    • hot flushes
    • sweats
  • Genital changes
    • dryness
    • frequency
  • psychological
    • memory
    • “Fog”

  • Decrease in estrogen production due to age linked decline in the number of ovarian follicles
  • Average age is around 51 years

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A 52-year-old female presents to the GP concerned she has passed through the menopause following no periods for 1 year. What is the cause of the end of the menstrual cycle?

  1. The decline in ovarian follicles
  2. Reduced production of GnRH
  3. Reduced production of FSH
  4. Reduced production of oestrogen
  5. Endometrial atrophy

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A 52-year-old female presents to the GP concerned she has passed through the menopause following no periods for 1 year. What is the cause of the end of the menstrual cycle?

  • The decline in ovarian follicles
  • Reduced production of GnRH
  • Reduced production of FSH
  • Reduced production of oestrogen
  • Endometrial atrophy

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A perimenopausal patient is given hormone replacement therapy (HRT) to alleviate their symptoms, including both an oestrogen and progesterone component. Their GP warns that HRT is associated with several complications. Which complication is this patient at an increased risk of developing?

  1. Atheroma formation
  2. Endometrial cancer
  3. Stroke
  4. Vaginal atrophy

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A perimenopausal patient is given hormone replacement therapy (HRT) to alleviate their symptoms, including both an oestrogen and progesterone component. Their GP warns that HRT is associated with several complications. Which complication is this patient at an increased risk of developing?

  • Atheroma formation
  • Endometrial cancer
  • Stroke
  • Vaginal atrophy

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Marge is a 52-year-old patient, presenting to you, her GP, with unpleasant menopausal symptoms lasting for over a year. These symptoms include hot flushes, headaches and fatigue. Her last period was 14 months ago, she has 3 grown-up children and has no surgical history. She demands to be prescribed an oestrogen-only HRT regimen as she has read on the internet that combined HRT is poorly tolerated. What is the appropriate thing to do here?

  1. Advice her to first try making lifestyle changes like reducing vasovagal triggers
  2. Tell her all patients get prescribed combined HRT
  3. Explain to her that oestrogen-only HRT should not be prescribed to patients with a uterus
  4. Explain to her that oestrogen-only HRT should not be prescribed to patients with a headache
  5. Advice her not to take HRT as cancer is its side effect

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Marge is a 52-year-old patient, presenting to you, her GP, with unpleasant menopausal symptoms lasting for over a year. These symptoms include hot flushes, headaches and fatigue. Her last period was 14 months ago, she has 3 grown-up children and has no surgical history. She demands to be prescribed an oestrogen-only HRT regimen as she has read on the internet that combined HRT is poorly tolerated. What is the appropriate thing to do here?

  • Advice her to first try making lifestyle changes like reducing vasovagal triggers
  • Tell her all patients get prescribed combined HRT
  • Explain to her that oestrogen-only HRT should not be prescribed to patients with a uterus
  • Explain to her that oestrogen-only HRT should not be prescribed to patients with a headache
  • Advice her not to take HRT as cancer is its side effect

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A 13-year-old girl is brought to the GP by her mother who is concerned about her lack of development. She is shorter than her peers, has no sign of breast growth, has no axillary or pubic hair, and has not started her periods. The girl's mother and grandmother both started their periods at age 14. The patient is otherwise healthy and takes no regular medications. How should this patient be managed?

  1. She requires investigation for primary amenorrhoea
  2. She should return to clinic if she has no sign of menstruation by age 15
  3. She requires investigation for secondary amenorrhoea

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A 13-year-old girl is brought to the GP by her mother who is concerned about her lack of development. She is shorter than her peers, has no sign of breast growth, has no axillary or pubic hair, and has not started her periods. The girl's mother and grandmother both started their periods at age 14. The patient is otherwise healthy and takes no regular medications. How should this patient be managed?

  1. She requires investigation for primary amenorrhoea
  2. She should return to clinic if she has no sign of menstruation by age 15
  3. She requires investigation for secondary amenorrhoea

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6 year old girl, with 6 months history of pubic hair growth associated with fine axillary hair as well as adult odor to sweat. No breast development with no acceleration of growth. Otherwise normal history and examinations. What is the most likely diagnosis?

  1. Precocious puberty
  2. Premature adrenarche
  3. Premature thelarche

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6 year old girl, with 6 months history of pubic hair growth associated with fine axillary hair as well as adult odor to sweat. No breast development with no acceleration of growth. Otherwise normal history and examinations. What is the most likely diagnosis?

  1. Precocious puberty
  2. Premature adrenarche
  3. Premature thelarche

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Milestone

Definition

Typical Age Range

Adrenarche

Activation of adrenal glands, leading to androgen production

6–8 years

Thelarche

Breast development due to estrogen

8–13 years (average 10-11)

Pubarche

Development of pubic and axillary hair due to androgens

8–14 years (often after adrenarche)

Menarche

First menstrual period

11–14 years (average 12-13)

Puberty

General development of secondary sexual characteristics and growth spurts

8–14 years (typically earlier in girls)

Testicular Enlargement (in boys)

Increase in testicle size due to testosterone production

9–14 years

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Puberty

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Neuroscience Revision

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Key areas- Neuro

  • Motor pathways – UMN and LMN
  • Sensory pathways
  • Epilepsy
  • Movement disorders (Parkinson’s disease)
  • Neuroinflammatory disorders (MS, meningitis)
  • Stroke and blood supply of the brain
  • Headache types
  • Dementia

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Thinking about localising pathology…

  • THINK - WHERE, WHEN, WHY

WHERE?

  • MOTOR- UMN, LMN, NMJ, MUSCLE
  • SENSORY- BRAIN, CORD, NERVE ROOT, NERVE
  • SYMPATHETIC/PARASYMPATHETIC 

WHEN?

  • INSTANTANEOUS - VASCULAR, EPILEPTIC
  • MINUTES- MIGRAINE
  • HOURS/DAYS - INFECTIVE, INFLAMMATORY
  • YEARS- NEURODEGENERATIVE

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A 73 y/o man presents to A&E with a left-sided facial droop and slurred speech. His cranial nerves are assessed-�* Unable to smile on left side�* Can close both eyes�Can raise both eyebrows symmetrically and wrinkle his forehead��Where is the lesion located? ��

  1. Left UMN
  2. Temporal branch of the facial nerve (CN7)
  3. Right UMN
  4. Right LMN
  5. Left LMN

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A 73 y/o man presents to A&E with a left-sided facial droop and slurred speech. His cranial nerves are assessed-�* Unable to smile on left side�* Can close both eyes�Can raise both eyebrows symmetrically and wrinkle his forehead��Where is the lesion located? ��

  1. Left UMN
  2. Temporal branch of the facial nerve (CN7)
  3. Right UMN
  4. Right LMN
  5. Left LMN

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A 43 y/o female patient comes in to ward complaining of muscle weakness. On investigation you find that her reflexes are greater and have increased tone (spasticity). What is a likely diagnosis?

  1. Motor neurone disease affecting both UMN and LMN
  2. LMN disorder
  3. Guillain-Barre syndrome
  4. Carpal tunnel syndrome
  5. UMN disorder

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A 43 y/o female patient comes in to ward complaining of muscle weakness. On investigation you find that her reflexes are greater and have increased tone (spasticity). What is a likely diagnosis?

  1. Motor neurone disease affecting both UMN and LMN
  2. LMN disorder
  3. Guillain-Barre syndrome
  4. Carpal tunnel syndrome
  5. UMN disorder

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Distinguishing between UMN and LMN lesions

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What type of lesion does this represent?

  1. Occlusion of the right anterior cerebral artery
  2. Right-sided hemisection of the spinal cord
  3. Left-sided hemisection of the spinal cord
  4. Parkinson's disease
  5. Complete transection of the spinal cord

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What type of lesion does this represent?

  1. Occlusion of the right anterior cerebral artery
  2. Right-sided hemisection of the spinal cord
  3. Left-sided hemisection of the spinal cord
  4. Parkinson's disease
  5. Complete transection of the spinal cord

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Sensory pathways/ ascending tracts

  • 3 neurones

  • Dorsal column pathway
  • Proprioception, vibration, fine touch
  • Decussate in the medulla

  • Spinothalamic pathway
  • Pain, temperature
  • Decussate immediately in the spinal column

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A patient presents with a 4 month history of progressive muscle weakness and numbness in the lower limbs. Investigations showed the presence of anaemia and vit B12 deficiency. The results of their neurological exam are given. Which area of the spinal cord is most likely to be affected?

  1. Dorsal column
  2. Ventral horn
  3. Anterior and lateral columns
  4. Anterior and dorsal columns

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A patient presents with a 4 month history of progressive muscle weakness and numbness in the lower limbs. Investigations showed the presence of anaemia and vit B12 deficiency. The results of their neurological exam are given. Which area of the spinal cord is most likely to be affected?

  1. Dorsal column
  2. Ventral horn
  3. Anterior and lateral columns
  4. Anterior and dorsal columns

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A cyclist falls off their bike during an accident. They come into clinic post the incident complaining of pain and paraesthesia in their outer thigh (lateral). Which spinal nerve root is likely to be affected?

  1. C5-C6
  2. S4-S5
  3. L2-L3
  4. T12

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A cyclist falls off their bike during an accident. They come into clinic post the incident complaining of pain in their outer thigh (lateral). Which spinal nerve root is likely to be affected?

  1. C5-C6
  2. S4-S5
  3. L2-L3
  4. T12

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A 45-year-old woman presents with worsening diplopia throughout the day. She has been suffering from this double vision for several weeks, and always feels that it is worse in the evenings and never present in the morning. On further questioning the patient reveals she believes her double vision improves after 'resting her eyes’. What is the most likely diagnosis?

  1. Migraine
  2. Diabetic neuropathy
  3. Myasthenia gravis
  4. Multiple sclerosis
  5. Guillain-Barre syndrome

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A 45-year-old woman presents with worsening diplopia throughout the day. She has been suffering from this double vision for several weeks, and always feels that it is worse in the evenings and never present in the morning. On further questioning the patient reveals she believes her double vision improves after 'resting her eyes’. What is the most likely diagnosis?

  1. Migraine
  2. Diabetic neuropathy
  3. Myasthenia gravis
  4. Multiple sclerosis
  5. Guillain-Barre syndrome

Bonus Q! How could you treat?

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A 45-year-old woman presents with worsening diplopia throughout the day. She has been suffering from this double vision for several weeks, and always feels that it is worse in the evenings and never present in the morning. On further questioning the patient reveals she believes her double vision improves after 'resting her eyes’. What is the most likely diagnosis?

  • Migraine
  • Diabetic neuropathy
  • Myasthenia gravis
  • Multiple sclerosis
  • Guillain-Barre syndrome

Bonus Q! How could you treat?

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A 27 y/o male presents with visual impairment, headaches and unexplained tiredness. His MRI shows a pituitary tumour compressing the optic chiasm. Which image would best represent the likely visual impairment this patient has?���

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A 27 y/o male presents with visual impairment, headaches and unexplained tiredness. His MRI shows a pituitary tumour compressing the optic chiasm. Which image would best represent the likely visual impairment this patient has?���

Bitemporal hemianopia

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A 27 y/o female involved in a car accident sustains a head injury. Her CT scan demonstrates a haemorrhage which is likely to be caused by damage to the bridging veins between the cortex and cavernous sinuses. What type of haemorrhage is this?

  1. Subarachnoid haemorrhage
  2. Extradural haemorrhage
  3. Intraparenchymal haemorrhage
  4. Subdural haemorrhage
  5. Intracerebellar haemorrhage

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A 27 y/o female involved in a car accident sustains a head injury. Her CT scan demonstrates a haemorrhage which is likely to be caused by damage to the bridging veins between the cortex and cavernous sinuses. What type of haemorrhage is this?

  1. Subarachnoid haemorrhage
  2. Extradural haemorrhage
  3. Intraparenchymal haemorrhage
  4. Subdural haemorrhage
  5. Intracerebellar haemorrhage

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A 70-year-old man presents to the clinic with a one-year history of progressive difficulty walking and occasional trembling in his hands. He reports feeling stiff, especially in the morning, and notes that he struggles to keep up with his grandchildren when walking. His wife mentions that his handwriting has become smaller and less legible. On physical examination, the patient demonstrates mild rigidity in the upper limbs, reduced arm swing while walking, and slowness in rapid alternating hand movements. His reflexes are normal, and there is no muscle weakness.

Which of the following findings is most consistent with the likely underlying condition?

  1. Hyperreflexia and spasticity
  2. Postural instability and asymmetric rigidity
  3. Severe weakness with fasciculation
  4. Intention tremor and dysmetria
  5. Wide-based gait with ataxia

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A 70-year-old man presents to the clinic with a one-year history of progressive difficulty walking and occasional trembling in his hands. He reports feeling stiff, especially in the morning, and notes that he struggles to keep up with his grandchildren when walking. His wife mentions that his handwriting has become smaller and less legible. On physical examination, the patient demonstrates mild rigidity in the upper limbs, reduced arm swing while walking, and slowness in rapid alternating hand movements. His reflexes are normal, and there is no muscle weakness.

Which of the following findings is most consistent with the likely underlying condition?

  • Hyperreflexia and spasticity
  • Postural instability and asymmetric rigidity
  • Severe weakness with fasciculation
  • Intention tremor and dysmetria
  • Wide-based gait with ataxia

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Parkinson’s Disease

  • Progressive neurodegenerative disorder due to degeneration of dopaminergic neurons in the substantia nigra.
  • 2x more common in males
  • Later onset
  • abnormal accumulation and aggregation of α-Syn in the form of Lewy bodies

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You are on placement in intensive care. A 31 y/o man is admitted with closed head injuries after a road accident. His GCS is 14/15 with no focal neurological deficit. HR, BP and ICP are normal. He is is administered maintenance IV fluids. ��Few hours later- he becomes agitated and confused and his GCS is 11/15. HR= 101 bpm, BP= 161/89 mmHg and ICP is 18 mmHg (normal: 7–15 mm Hg). ��Which of the following pathophysiological changes explain his clinical deterioration?�

  1. Rise in ICP causing fall in cerebral perfusion pressure
  2. Fall in ICP causing rise in cerebral perfusion pressure
  3. Fall in cardiac output causing cerebral hypoxia
  4. Fall in blood volume causing a fall in cardiac output

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You are on placement in intensive care. A 31 y/o man is admitted with closed head injuries after a road accident. His GCS is 14/15 with no focal neurological deficit. HR, BP and ICP are normal. He is is administered maintenance IV fluids. ��Few hours later- he becomes agitated and confused and his GCS is 11/15. HR= 101 bpm, BP= 161/89 mmHg and ICP is 18mmHg (normal: 7–15 mm Hg). ��Which of the following pathophysiological changes explain his clinical deterioration?�

  1. Rise in ICP causing fall in cerebral perfusion pressure
  2. Fall in ICP causing rise in cerebral perfusion pressure
  3. Fall in cardiac output causing cerebral hypoxia
  4. Fall in blood volume causing a fall in cardiac output

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A middle-aged man presents with pain and numbness in the thumb, index finger and middle finger of his right hand. Which nerve roots are likely to be affected?

  1. C8-T1
  2. C5-T1
  3. T11-L4
  4. C3-C5
  5. T1-T6

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A middle-aged man presents with pain and numbness in the thumb, index finger and middle finger of his right hand. Which nerve roots are likely to be affected?

  1. C8-T1
  2. C5-T1
  3. T11-L4
  4. C3-C5
  5. T1-T6

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A 7 y/o girl is brought to the GP by her father after a few recurrent episodes of sudden loss of awareness. The father says that she “suddenly went blank and did not respond for a period of 10-15 seconds and then went back to normal.” She does not recall this and things nothing has happened. Her father also recalls light lip-smacking during these episodes. ��What is the most likely diagnosis?

  1. Juvenile myoclonic epilepsy
  2. Occipital lobe epilepsy
  3. Frontal lobe epilepsy
  4. Childhood absence epilepsy

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A 7 y/o girl is brought to the GP by her father after a few recurrent episodes of sudden loss of awareness. The father says that she “suddenly went blank and did not respond for a period of 10-15 seconds and then went back to normal.” She does not recall this and things nothing has happened. Her father also recalls light lip-smacking during these episodes. ��What is the most likely diagnosis?

  1. Juvenile myoclonic epilepsy
  2. Occipital lobe epilepsy
  3. Frontal lobe epilepsy
  4. Childhood absence epilepsy

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Epilepsy

  • Recurrent and spontaneous seizures
  • Seizure = abnormal firing of neurones in part(s) of the brain
  • Diagnosis: By history and eyewitness account.
  • pre-ictal signs (aura), post-ictal signs
  • Triggers
  • Risk factors: FHx, meningitis etc.

  • EEGs are useful to spot genetic epilepsies
  • Treatment: AEDs, surgery to remove a small part of the brain that's causing the seizures etc.
  • Common types of AED include (NHS):
  • sodium valproate
  • Carbamazepine
  • Lamotrigine

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A 17 y/o boy has been in status epilepticus for 45 mins. He is given IV lorazepam before which is a benzodiazepine. His seizures stop and he regains consciousness. How does this class of drugs work in seizure treatment?

  1. Increasing acetylcholine binding to muscarinic receptors
  2. Facilitating binding of GABA at its receptors in the CNS
  3. Blocks reabsorption of serotonin into neurons
  4. inhibition of neurotransmitter release from the primary afferent terminals in the spinal cord

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A 17 y/o boy has been in status epilepticus for 45 mins. He is given IV lorazepam before which is a benzodiazepine. His seizures stop and he regains consciousness. How does this class of drugs work in seizure treatment?

  1. Prevent breakdown of acetylcholine
  2. Facilitating binding of GABA at its receptors in the CNS
  3. Blocks reabsorption of serotonin into neurons
  4. inhibition of neurotransmitter release from the primary afferent terminals in the spinal cord

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Very short answer question:��A 78 y/o woman presents with 12 hr Hx of severe, persistent headache. She describes this as the worst headache she has had and says it is worst at the back of her head. ��She has no history of trauma and her no other signs and symptoms. She has PMH of hypertension for which she is taking amlodipine. ��What is the most like diagnosis?

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Very short answer question:��A 78 y/o woman presents with 12 hr Hx of severe, persistent headache. She describes this as the worst headache she has had and says it is worst at the back of her head. ��She has no history of trauma and her no other signs and symptoms. She has PMH of hypertension for which she is taking amlodipine. ��What is the most like diagnosis?��Subarachnoid haemorrhage

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A 25 y/o woman comes to the GP complaining of a headache occurring once every few weeks for the past 6 months. She describes seeing flashing lights and needs to lie down in a dark room for it to pass. She also mentions that it normally occur on the RHS. What is the most likely diagnosis?

  1. Tension headache
  2. Focal epilepsy
  3. Cluster headache
  4. Subarachnoid haemorrhage
  5. Migraine

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A 25 y/o woman comes to the GP complaining of a headache occurring once every few weeks for the past 6 months. She describes seeing flashing lights and needs to lie down in a dark room for it to pass. She also mentions that it normally occur on the RHS. What is the most likely diagnosis?

  1. Tension headache
  2. Focal epilepsy
  3. Cluster headache
  4. Subarachnoid haemorrhage
  5. Migraine

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An 81 y/o man presents to A&E with sudden onset weakness in his left leg. On examination there is hyperreflexia in the ankles. Which artery is most likely to be occluded?

  1. R anterior cerebral artery
  2. L middle cerebral artery
  3. L carotid artery
  4. R middle cerebral artery
  5. Left posterior cerebral artery

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An 81 y/o man presents to A&E with sudden onset weakness in his left leg. On examination there is hyperreflexia in the ankles. Which artery is most likely to be occluded?

  1. R anterior cerebral artery
  2. L middle cerebral artery
  3. L carotid artery
  4. R middle cerebral artery
  5. Left posterior cerebral artery

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Which of these statements is false?�

  1. Cranial nerves IX-XII arise from the medulla
  2. The facial nerve controls lacrimation
  3. Cranial nerve IV has purely motor functions
  4. Cranial nerve I is responsible for pupillary contraction
  5. All of the above

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Which of these statements is false?�

  1. Cranial nerves IX-XII arise from the medulla
  2. The facial nerve controls lacrimation
  3. Cranial nerve IV has purely motor functions
  4. Cranial nerve I is responsible for pupillary contraction
  5. All of the above

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A young woman presents after multiple episodes of optic neuritis, during which she develops unilateral eye pain. Upon examination, she is found to have decreased visual acuity and colour saturation on her affected eye. Her doctor suspects multiple sclerosis. ��Which features would be expected on a T2-weighted MRI?

  1. Single hyperintense lesion
  2. Midline shift
  3. Many hyperintense lesions
  4. Cortical atrophy

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A young woman presents after multiple episodes of optic neuritis, during which she develops unilateral eye pain. Upon examination, she is found to have decreased visual acuity and colour saturation on her affected eye. Her doctor suspects multiple sclerosis. ��Which features would be expected on a T2-weighted MRI?

  1. Single hyperintense lesion
  2. Midline shift
  3. Many hyperintense lesions
  4. Cortical atrophy

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Multiple sclerosis

  • Chronic, autoimmune, inflammatory and degenerative disease of the central nervous system
  • Inflammation, demyelination, neuronal injury and loss, scarring
  • Can be relapsing-remitting, primary progressive or secondary progressive
  • Diagnosis: dissemination in SPACE and TIME; McDonalds criteria
  • MRI and lumbar puncture

  • Relapses treated with immunomodulatory and immunosuppressive therapies

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A young man presents with the inability to produce coherent sentences after a right middle cerebral artery infarct. He puts in a lot of effort to finish his sentences but struggles to do so. His understanding of spoken language is intact. The GP suspects neurological damage from the infarct. ��Which part of the brain has likely been affected?

  1. Wernicke’s area
  2. Frontal and occipital lobes of the left hemisphere
  3. Right superior temporal gyrus
  4. Occipital lobe of the right hemisphere
  5. Broca’s area

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A young man presents with the inability to produce coherent sentences after a right middle cerebral artery infarct. He puts in a lot of effort to finish his sentences but struggles to do so. His understanding of spoken language is intact. The GP suspects neurological damage from the infarct. ��Which part of the brain has likely been affected?

  1. Wernicke’s area
  2. Frontal and occipital lobes of the left hemisphere
  3. Right superior temporal gyrus
  4. Occipital lobe of the right hemisphere
  5. Broca’s area

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A 6 year old girl presents with a non-blanching rash and neck stiffness over the last 6 hours. She describes headache that is worsened in bright rooms and has a fever of 38.1C. Give the likely diagnosis and the best diagnostic test?�

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A 6 year old girl presents with a non-blanching rash and neck stiffness over the last 6 hours. She describes headache that is worsened in bright rooms and has a fever of 38.1C. Give the likely diagnosis and the best diagnostic test?�

Bacterial meningitis

Lumbar puncture

Neutrophils

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Further resources

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