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In vitro generation of functional murine heart organoids via FGF4 and extracellular matrix

Lee, J., Sutani, A., Kaneko, R., Takeuchi, J., Sasano, T., Kohda, T., Ihara, K., Takahashi, K., Yamazoe, M., Morio, T., Furukawa, T., & Ishino, F.

Nature Communications (2020)

Mark Ghaly, Annabelle Nettey, and Tanyaradzwa Makunike

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Background Information

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Cardiogenesis

(Chong et al., 2014)

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Embryonic Mouse Heart Development

(Lee et al., 2020)

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(Sigma Aldrich, n.d.)

FGF Superfamily

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(Sigma Aldrich, n.d.)

FGF Superfamily

FGF4

FGF10

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Laminin-Entactin (LN/ET) Complex

(Arends & Lieleg, 2016)

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Heart Organoid: the “Mini Heart”

(Betts et al., 2013; Hastings, 2021)

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Research Goal

Generate a heart organoid similar to an embryonic mouse heart to determine key components of cardiogenesis

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Hypothesis

The presence of fibroblast growth factor 4 (FGF4) and the laminin-entactin (LN/ET) complex is crucial for cardiogenic differentiation and heart formation

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Methods

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Isolating Embryonic Stem Cells

Formation of Embryonic Bodies

+ LIF

In vitro cardiogenesis, adding:

Experimental Design — LN/ET complex

LN/ET complex + FGF4

Matrigel

complex + FGF4

FGF4 only

(Control)

Cultured samples for 10-15 days

Analyzing morphological changes + heart organoid beat (bpm)

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Isolating Embryonic Stem Cells

Formation of Embryonic Bodies

+ LIF

In vitro cardiogenesis; adding LN/ET complex and either:

Experimental Design — FGF Signals

FGF`10

FGF4

Cultured samples for 10-15 days

Analyzing heart organoid formation efficiency (%)

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Results

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Embryonic Murine Heart Development

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Heart organoid development in embryonic bodies cultured in LN/ET complex + FGF4

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Morphological changes and beating capacity were observed at day 3 and increased by day 6

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Heart organoid growth in embryoid bodies cultured in Matrigel is constrained vs. LN/ET complex

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Heart organoid generation is less

efficient in embryonic bodies cultured in

LN/ET + FGF10 compared to LN/ET + FGF4

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Heart organoid generation is less

efficient in embryonic bodies cultured in

LN/ET + FGF10 compared to LN/ET + FGF4

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Conclusion

LN/ET complex and FGF4 are KEY components of embryonic heart development!

In vitro cardiogenesis of embryonic bodies cultured in a FGF4 and LN/ET complex mimics in vivo cardiogenesis in mice.

i.e. the authors’ hypothesis was supported

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Future Studies

  • How long are FGF4 + LN/ET complex needed?
    • Consequences of inhibition at various stages of development?
  • Heart organoid + brain organoid = ?
  • Why are heartbeats occurring?
    • Origin of signal

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References

Aisenbrey, E. & Murphy, W.L. (2020, May 27). Synthetic alternatives to Matrigel. Nature Reviews Materials, 5, 539-551. https://doi.org/10.

1038/s41578-020-0199-8

Arends, F. & Lieleg, O. (2016). Biophysical properties of the basal lamina: A highly selective extracellular matrix. In Francesco Travascio (Ed.),

Composition and function of the extracellular matrix in the human body. IntechOpen. https://doi.org/10.5772/62519

Betts, J.G., Young, K.A., Wise, J.A., Johnson, E., Poe, B., Kruse, D.H., Korol, O., Johnson, J.E., Womble, M., & DeSaix, P. (2013, April 25). An

Introduction to the Human Body. Anatomy and Physiology. OpenStax. https://openstax.org/books/anatomy-and-physiology/

pages/1-introduction

Chong, J.J.H., Forte, E., & Harvey, R.P. (2014, September 26). Developmental origins and lineage descendants of endogenous adult cardiac

progenitor cells. Stem Cell Research, 13, 592-614. http://dx.doi.org/10.1016/j.scr.2014.09.008

Hastings, C. (2021, May 25). Cardiac organoids self-organize to mimic human heart. Medgadget. https://www.medgadget.com/

2021/05/cardiac-organoids-self-organize-to-mimic-human-heart.html

Sigma Aldrich. (n.d.). Fibroblast growth factor family (FGF). https://www.sigmaaldrich.com/CA/en/technical-documents/technical-article/

cell-culture-and-cell-culture-analysis/mammalian-cell-culture/fibroblast-growth

Wojciechowska, A., Braniewska, A., & Kozar-Kaminska, K. (2017, April 27). MicroRNA in cardiovascular biology and disease. Advances in Clinical

and Experimental Medicine, 26(5), 865-874.10.17219/acem/62915

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