CYRIAX TECHNIQUE

DR.MAHENDRAN

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• 01.Question: According to Cyriax's pathophysiological model, what is the fundamental pathological change in tendinopathy that DTFM primarily aims to address?
• A) Inflammatory infiltration of neutrophils and macrophages
• B) Disorganized collagen deposition with loss of parallel fiber alignment 
• C) Complete avascular necrosis of the tendon core
• D) Hypertrophic thickening of the tendon sheath

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• 02.Question: In selective tissue tension testing, what clinical finding differentiates a muscle strain from a tendinopathy?
• A) Pain only on passive elongation of the muscle
• B) Pain on active contraction against resistance, with less pain on passive stretch 
• C) Pain on both active and passive movements equally
• D) Complete absence of pain on any movement

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• 03.Question: Recent systematic reviews on Deep Transverse Friction Massage have concluded that:
• A) DTFM is superior to all other manual therapy techniques
• B) Evidence is limited and conflicting; DTFM is not superior to placebo for most tendinopathies 
• C) DTFM should be the first-line treatment for all tendon pathologies
• D) DTFM is contraindicated in all chronic conditions
• 04.Question: In the Cyriax approach, Mill's manipulation for lateral epicondylitis involves which specific movement sequence?
• A) Elbow flexion with wrist flexion and pronation
• B) Elbow extension with wrist flexion and forearm pronation 
• C) Elbow flexion with wrist extension and supination
• D) Elbow extension with wrist extension and supination

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• 05.Question: Which of the following is a critical contraindication to the Cyriax friction massage technique that requires careful differential diagnosis?
• A) Chronic lateral epicondylitis
• B) Coagulopathy or anticoagulant therapy 
• C) Postural muscle tightness
• D) Scar tissue formation > 6 months old

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• Question: In Cyriax's orthopedic medicine framework, the "lesion" most amenable to DTFM is characterized by which histological feature?
• A) Fibroblastic proliferation with collagen disorganization and neovascularization 
• B) Acute hemorrhagic infiltration
• C) Complete tendon rupture with gap formation
• D) Atrophic degeneration with fatty infiltration

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