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Management of intestinal obstruction

Dr Shehu H�Lecturer/Paediatric Surgeon

Bhuth, Jos.

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Outline

  • Introduction
  • Classifications
  • Aetiology
  • Pathophysiology
  • Clinical presentations
  • Management
  • Complications
  • Conclusion

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Introduction

Definition

  • Failure of cranio-caudal movement of bowel contents due to an impediment

  • May also result from functional impairment without mechanical obstruction

  • It is a common medical emergency occurring worldwide. Account for 5% of acute surgical admissions

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Introduction

  • It is commoner in the small bowel than large bowel ( 80% )

  • Early diagnosis is important and correct diagnosis even better

  • Treatment should be prompt and appropriate as mortality with strangulation is up to 30%

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CLASSIFICATION

● DYNAMIC:- Where peristalsis is working against a mechanical obstruction.

According to the site of obstruction it is further classified into

♦ Intraluminal

♦ Intramural

♦ Extramural

● ADYNAMIC:- It may occur in two forms:-

♦ Peristalsis absent ( e.g. Paralytic ileus )

♦ Peristalsis present in non-propulsive form(e.g. Mesenteric

vascular occlusion)

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Adynamic

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Causes

Metabolic

Post. Operative

ileus

Neuropathic

disorders

Retroperitoneal

process

Diffuse

peritonitis

Response to

localized

Inflammatory

process

Medications

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DYNAMIC

INTRALUMINAL

♦Fecal impaction

♦Foreign bodies

♦Trico bezoar

♦ Gallstones

Trico bezoar

Gallstones ileus

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INTRAMURAL

♦ Stricture(tuberculous stricture)

♦ Malignancy

♦ Crohn’s Disease

EXTRAMURAL

♦ Bands /

Adhesions(40%)

♦ Hernia(25%)

♦ Volvulus

♦ Intussuception

Crohn’s disease

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CLASSIFICATION

CONGENITAL

ACQUIRED

♦ Anorectal malformations

♦ Congenital megacolon

♦ Duodenal atresia

♦ Volvulus

♦ Bands and adhesions

♦ Intestinal atresia(ileal)

♦ Hernia(commonest)

♦ Postoperative

♦ Intussusceptions

♦ Gallstones

♦ Tuberculosis

♦ Malignancy

♦ Roundworm

Intussusceptions

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CLASSIFICATION :- Depending on site of obstruction

Proximal Small Bowel(Duodenum and jejunum)

Distal Small Bowel (Ileum)

Large Bowel

♦ Congenital

♦ Lipomas

♦ Malignancy

♦ Bands and Adhesions

♦ Hernias- Common

cause

♦ Malignancy

♦ Crohn’s Disease

♦ Tuberculosis

strictures

♦ Malignancy

♦ Tuberculous

strictures

♦ Anorectal malformation

♦ Volvulus

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CLASSIFICATION :- According to Pathological Changes

  • Simple – only a single limb is involved

  • Strangulated – where there is direct interference to

blood flow

  • Closed loop obstruction – when bowel obstructed

both at proximal and distal points

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�����Aetiology

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Hernia(EXTERNAL/INTERNAL)

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ADHESIONS

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VOLVOLUS

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INTUSSUSCEPTION

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ASCARIASIS

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FOREIGN BODY

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TUMOURS

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INTESTINAL ATRESIAS

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DUODENAL WEB

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CROHN’S DISEASE

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DIVERTICULITIS

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COMMON CAUSES OF INTESTINAL OBSTRUCTION ACCORDING TO AGE

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����Pathophysiology

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Intra abdominal surgery

Motility post operation is as follows:

  • Small bowel within 24-48 hours

  • Gastric within 48hours

  • Colonic within 3-5 days

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Simple obstruction

  • Below the obstruction
  • Normal peristalsis and absorption
  • Becomes empty and collapse

Contracted and becomes immobile

  • Constipation

  • Spurious diarrhea

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Simple obstruction

  • Above the obstruction
  • Increasing peristalsis
  • If this fail bowel distension occurs
  • Resulting in flaccidity and paralysis

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Simple obstruction

  • The gases are mostly swallowed

  • Also from products of putrefaction

  • Normal intestinal digestive juices ( 8-10 Liters/24 hours)

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Simple obstruction

  • Accumulation of fluid occurs proximally

  • Hypovolemia and dyselectrolytaemia

  • Raised intraluminal pressure

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Simple obstruction

  • The lymphatic and venous pressure are exceeded first
  • Venous congestion
  • Edema of wall occur
  • Movement of fluid from plasma into gut lumen and peritoneal cavity
  • Death occur in simple intestinal obstruction from hypovolemia and dyselectrolytaemia

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Simple obstruction

  • The higher the level of obstruction, the earlier the onset of vomiting and fluid and electrolyte imbalance

  • In high obstruction, metabolic alkalosis results because acid rich fluid is lost

  • In low obstruction, metabolic acidosis result as the sequestered fluid is alkaline

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Strangulated obstruction

  • When the pressure of the occluding band exceeds venous pressure

  • Venous engorgement of gut wall occurs

  • Dilatation of intramural lymph channels that carry multiplying bacteria from mucosa into systemic circulation

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Strangulated obstruction

  • Translocation of bacteria causes endotoxaemia

  • Increase venous pressure progressed to rupture of capillaries and then bleeding into lumen, wall or peritoneal cavity

  • Arterial compromise cause necrosis and perforation

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Closed Loop Obstruction

  • Afferent & efferent limbs of bowel are obstructed

  • Typically seen in external hernias

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Closed loop obstruction

  • Seen also in colonic obstruction with competent iliocaecal valve

  • The rich bacterial floral adds to the production of gases

  • Rapid distension ► ↑luminal pressure ► circulation impairment ► bowel necrosis & perforation ► fulminant peritonitis.

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Closed loop obstruction

  • Types of Loop obstruction
  • Type 1
  • A- colonic obstruction with competent iliocaecal valve
  • B- colonic obstruction with very competent iliocaecal valve
  • Type 2
  • Colonic obstruction with incompetent iliocaecal valve

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���� Clinical Presentation

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Clinical presentation

The cardinal features of obstruction are pain, vomiting, distension & constipation but clinical presentation varies according to:

    • Site of obstruction
    • Duration before Presentation
    • Underlying pathology
    • The presence or absence of ischemia

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Clinical presentation

  1. Abdominal pain
  2. Usually 1st
  3. Colicky, intermittent
  4. In SBO central, waxes rapidly

But wanes slowly

  • Last 3-5 minutes
  • May persist in between

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Clinical presentation

  1. Abdominal pain
  2. LBO is Cramping
  3. Lower abdomen/ flank
  4. Last 15- 30 minutes
  5. Pain may be absent

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Clinical presentation

2. Abdominal distension

  • The lower the site of obstruction the more the distension.

  • It varies inversely as the vomiting.

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Clinical presentation

2. Abdominal distension

  • Central in small bowel obstruction.

  • More in the flanks in colonic obstruction

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Clinical presentation

3. Vomiting

  • In High SBO- bilious & early
  • In Low SBO- feculent
  • In LBO- delayed and feculent

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Clinical presentation

4. Constipation

  • Occurs Early in “lower” Large Bowel Obstruction.

  • Occurs Late in

“High” Small Bowel Obstruction.

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���� Examination findings

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Examination Findings

  • Dehydration

Common in small bowel obstruction

Vomiting and fluid sequestration

Pyrexia

may indicate:

• the onset of ischemia

• intestinal perforation

• inflammation associated with the obstructing disease

Hypothermia indicates septicemic shock

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Clinical examination

  • Incarcerated hernias may be obscure (obese)
  • Surgical scars can suggest adhesions
  • Palpation abdominal masses can suggest neoplasms, intussusception, abscess
  • Abdominal auscultation period of increasing separated by periods of quite bowel sounds (high pitched, tinkling or musical) in mechanical obstruction
  • Rectal examination to seek luminal masses. Blood in the feces suggest mucosal lesion (cancer, intussusception, infarction)

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���Investigations

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Radiological features of plain X - Ray

  • Small bowel – straight segments generally central and lie transversely
  • Jejunum – valvulae conniventes, spaced regularly, concertina or ladder effect
  • Ileum – characteristically unremarkable
  • Caecum – rounded gas shadow in right iliac fossa
  • Large bowel except caecum – haustral folds, spaced irregularly

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Erect abdominal X-ray

  • Upright view of abdomen in a patient with intestinal obstruction,
  • Showing multiple air fluid levels

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Supine abdominal X-ray

  • Dilated loops of small bowel are visible(arrows)
  • Transversely oriented
  • Valvulae conniventes

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Lateral decubitus abdominal X-ray

  • Showing air fluid levels consistent with intestinal obstruction (arrows)

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Barium follow - through�Following features may assist in diagnosis

  • Delay in the transit time
  • Clawhand appearance
  • Beak sign
  • Springcoil sign
  • Friedman Dhal sign

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Barium meal

  • Jejunojejunal Intussusception

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CT Scan

  • Rt colonic tumour

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Ultrasonography

  • It is of particular value in looking at the dynamics of the small bowel
  • Used to assess peristalsis

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Other Investigations

  • CBC
  • Group & crossmatch blood
  • Urea and Electrolyte
  • RBS

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����� Treatment

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Aim of Rx

Aim is to relieve obstruction as soon as possible before strangulation occurs or before systemic complications set in

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Resuscitation

  • Nil per oral/ Nasogastric tube
  • Fluid and electrolyte imbalance correction
  • Urethral catheterization
  • Antibiotics
  • Analgesics
  • Correct anaemia

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Conservative treatment

  • Partial obstruction
  • Early post op obstruction
  • Obstruction secondary to Crohn’s disease
  • Recurrent obstruction

Open surgery if no improvement after 24hrs

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Operative Treatment

  • Procedure depends on cause of obstruction
  • Non-viable gut must be resected
  • Questionable gut should be checked for viability

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Surgery types

  • Resection- The diseased part of the small intestine (ileum) is removed. The two healthy ends are then sewn back together and the incision is closed.

Indications

  • Gangrenous bowel

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Surgeries

  • In cases of strangulated Inguinal/femoral hernias the standard groin incision is given & the weakness repaired using hernioplasty or herniorrhaphy, with bowel resection if required.

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Surgeries

  • In adhesive obstructed cases, laproscopic adhesiolysis (adhesive band lysis) is carried out

  • Bypass: Anastomosis of proximal small bowel or large intestine distal to the obstruction may be a good procedure in some cases of carcinoma or radiation injury

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Specific Rx

    • Hirshsprung's- pull through
    • Intestinal atresia- resection + anastomosis
    • Duodenal atresia- duodenoduodenostomy
    • Meconium ileus - resection + anastomosis

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Complications

  • Fluid and dyselectrolytaemia
  • Hypovolemic / Endotoxic Shock
  • Peritonitis
  • Adhesion/ Garres’ obstruction
  • Acute Renal Failure
  • Multiple organ failure

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Prognosis

  • Type of obstruction
  • Duration of obstruction
  • Cause of obstruction
  • Age of the patient
  • Length of gangrenous bowel

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“Never let the sun rise or set on small-bowel obstruction” �