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Labour & Delivery�Suwandi Dewapura�skdew4@student.monash.edu

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Today’s Lecture

  • Stages of labour
  • Instrumental & Operative deliveries
  • Birth trauma
  • Intrapartum fetal monitoring
  • Induction
  • Vaginal birth after Caesarean (VBAC)

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Not covered here:

  • Mechanism of labour
  • Physiology of labour
  • OSCE-style management of labour (Hx, Ex)
  • Analgesia in labour

  • Malpresentations (covered later today)

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Stages of labour

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Stage 1:

0 🡪 10cm dilation

Active

Latent

A quick word about Rupture of Membranes (ROM):

  • Rupture of amniotic sac
  • Usually spontaneous (SROM), in first stage of labour
  • However, can be delayed (during second stage)
  • Can also be premature (PROM/PPROM) – not covered here.

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Stage 1:

0 🡪 10cm dilation

  • Contractions are mild and infrequent
  • Cervical dilation occurs at a slower rate (<1cm/hr)
  • May last up to 14– 20 hours depending on parity

  • From 6cm to 10cm dilation
  • Rate of dilation increases from latent phase
  • (1.2-1.5 cms/hour)
  • Duration: 2-3 hrs in multis; 4-6 hrs in primips

Management of normal Stage 1:

  • Analgesia as required
  • Determine fetal position by examination
  • Assess cervical dilation & descent of fetal head regularly (VE 4 hourly)
  • +/- CTG
  • +/- Amniotomy during active phase (not necessary if labour progressing well)

Active

Latent

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Active

Latent

Stage 1:

0 🡪 10cm dilation

Stage 2:

Fully dilated to birth of infant

During this stage:

  • Regular contractions (increasing frequency & intensity)
  • Crowning (appearance of fetal head at vaginal opening with contractions)

Duration:

  • <2 hrs for primips
  • <1 hr for multis
  • (+1 hr if epidural analgesia)

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Active

Passive

Stage 2:

Fully dilated to birth of infant

Presenting part descends through the pelvis (no urge to push)

Maternal effort to drive presenting part through birth canal

During this stage:

  • Regular contractions (increasing frequency & intensity)
  • Crowning (appearance of fetal head at vaginal opening with contractions)

Duration:

  • <2 hrs for primips
  • <1 hr for multis
  • (+1 hr if epidural analgesia)

Active

Latent

Stage 1:

0 🡪 10cm dilation

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Active

Passive

Stage 2:

Fully dilated to birth of infant

Management of normal Stage 2:

  • Warm compresses, comfortable & safe positions
  • Episiotomy if required

(indications include shoulder dystocia, instrumental delivery or vaginal breech)

  • +/- Delay cord clamping at least 1 minute, unless evidence of compromise and need for neonatal resuscitation

Active

Latent

Stage 1:

0 🡪 10cm dilation

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Stage 3

Active

Passive

Stage 2:

Fully dilated to birth of infant

Stage 3:

Birth to expulsion of placenta

Active

Latent

Stage 1:

0 🡪 10cm dilation

During this stage:

  • Contractions to expel the placenta
  • Eventually, expulsion of placenta (usually with associated bleeding)

Duration:

  • 30 mins with Active Mx
  • 60 mins with Physiological Mx (expulsion by maternal effort only)

Management of normal Stage 3:

  • Fundal massage (induce contraction + stop bleeding)
  • Active management to reduce risk of PPH

10 units oxytocin IM

Controlled cord traction (placenta separate on its own)

  • Perform placental examination to ensure complete
  • Repair perineal lacerations, if any
  • Assess blood loss & manage appropriately

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Management of prolonged labour

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Stage

Management

Prolonged Stage 1 – Latent Phase

Conservative/expectant mx. Can consider ARM, cervical ripening or oxytocin.

Prolonged Stage 1 – Active Phase

Augment labour with oxytocin if weak contractions; analgesia if strong contractions.

+/- ARM, cervical ripening

Caesarean if above management ineffective.

Prolonged Stage 2

Oxytocin if weak contractions or <1cm progress after 60-90 mins pushing.

Trial of instrumental delivery if fetal head engaged & sufficient contractions.

Caesarean if head not engaged / insufficient contractions.

Prolonged Stage 3

Uterotonic agent, controlled cord traction, empty bladder.

IV access, FBE, Group & Hold.

Manual removal of placenta (MROP). Hysterectomy if failure of MROP.

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Management of prolonged labour

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Stage

Management

Prolonged Stage 1 – Latent Phase

Conservative/expectant mx. Can consider ARM, cervical ripening or oxytocin.

Prolonged Stage 1 – Active Phase

Augment labour with oxytocin if weak contractions; analgesia if strong contractions.

+/- ARM, cervical ripening

Caesarean if above management ineffective.

Prolonged Stage 2

Oxytocin if weak contractions or <1cm progress after 60-90 mins pushing.

Trial of instrumental delivery if fetal head engaged & sufficient contractions.

Caesarean if head not engaged / insufficient contractions.

Prolonged Stage 3

Uterotonic agent, controlled cord traction, empty bladder.

IV access, FBE, Group & Hold.

Manual removal of placenta (MROP). Hysterectomy if failure of MROP.

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Management of prolonged labour

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Stage

Management

Prolonged Stage 1 – Latent Phase

Conservative/expectant mx. Can consider ARM, cervical ripening or oxytocin.

Prolonged Stage 1 – Active Phase

Augment labour with oxytocin if weak contractions; analgesia if strong contractions.

+/- ARM, cervical ripening

Caesarean if above management ineffective.

Prolonged Stage 2

Oxytocin if weak contractions or <1cm progress after 60-90 mins pushing.

Trial of instrumental delivery if fetal head engaged & sufficient contractions.

Caesarean if head not engaged / insufficient contractions.

Prolonged Stage 3

Uterotonic agent, controlled cord traction, empty bladder.

IV access, FBE, Group & Hold.

Manual removal of placenta (MROP). Hysterectomy if failure of MROP.

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Management of prolonged labour

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Stage

Management

Prolonged Stage 1 – Latent Phase

Conservative/expectant mx. Can consider ARM, cervical ripening or oxytocin.

Prolonged Stage 1 – Active Phase

Augment labour with oxytocin if weak contractions; analgesia if strong contractions.

+/- ARM, cervical ripening

Caesarean if above management ineffective.

Prolonged Stage 2

Oxytocin if weak contractions or <1cm progress after 60-90 mins pushing.

Trial of instrumental delivery if fetal head engaged & sufficient contractions.

Caesarean if head not engaged / insufficient contractions.

Prolonged Stage 3

Uterotonic agent, controlled cord traction, empty bladder.

IV access, FBE, Group & Hold.

Manual removal of placenta (MROP). Hysterectomy if failure of MROP.

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Induction of labour

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Induction of Labour (IOL)

Common indications

  • Prolonged pregnancy
  • FGR
  • Pre-eclampsia & other hypertensive disorders
  • Deteriorating maternal illness
  • Diabetes mellitus
  • PROM

Contraindications

  • Hx of uterine rupture
  • Previous classical Caesarean or high-risk Caesarean
  • Vasa/placenta praevia
  • Malpresentation
  • Cord prolapse
  • Active genital herpes
  • Non-reassuring FHR
  • Any other contraindication to labour or vaginal birth

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Bishop’s Score

  • Used to assess favourability of the cervix & whether induction will be successful
  • Simplified Bishop’s Score: only looking at fetal station, cervical dilation and cervical effacement
  • Modified Bishop’s Score: looks at five criteria

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If Bishop’s ≥5, cervix favourable for vaginal delivery.

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Options for IOL

  • Stretch & sweep (stimulates prostaglandin release)
  • Cervical ripening
    • Foley or Cook catheters (put pressure on cervix)
    • Prostaglandins (e.g. Cervidil)
  • ARM (increases pressure of fetal head on cervix)
    • Only if cervix partially dilated & completely effaced, and fetal head well-applied
  • Synthetic oxytocin (Syntocinon)

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Complications of IOL with Syntocinon

  • Excessive uterine activity & hyperstimulation
  • Uterine rupture
  • Water intoxication with high dose regimen or prolonged periods of use (oxytocin has an ADH-like effect)

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Instrumental delivery

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Indications for Instrumental Delivery

  • Confirmed/suspected fetal compromise
  • Lack of advancement of presenting part & maternal tiredness
  • Shorten & reduce the effects of second stage of labour if there are concurrent medical conditions
    • E.g. CHF NYHA Class III or IV, hypertensive crisis, myasthenia gravis, spinal cord injury & risk of autonomic dysreflexia, proliferative retinopathy

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Prerequisites for instrumental delivery

  • Adequate uterine contractions
  • Fetal head not palpable abdominally
  • Station of presenting part below ischial spines
  • Knowledge of exact position of head
  • Vertex presentation
  • Cervix fully dilated, membranes ruptured
  • Adequate pelvis (no CPD)
  • Empty bladder

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Instrumental delivery

Vacuum

Forceps

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Instrumental delivery

Vacuum

Forceps

Both require:

  • Sufficiently large birth canal
  • Fully dilated cervix
  • An engaged fetal head
  • Adequate uterine contractions

Both can be used for management of:

  • Prolonged second stage
  • Non-reassuring CTG
  • In the instance of maternal exhaustion

Both contraindicated if:

  • Fetal bleeding disorder
  • Predisposition to fractures

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Instrumental delivery

Vacuum

Forceps

  • Can be used for breech presentation
  • Can use Neville-Barnes for face presentations

  • Less likely to sustain scalp injuries
  • Can’t dislodge during delivery

  • Requires more space than vacuum
  • Higher risk of perineal trauma / tears
  • Not used for breech presentation
  • Not for face presentations

  • Risk of scalp injuries (C/I before 34 weeks)
  • Can dislodge during delivery

  • Doesn’t require much space
  • Lower risk of perineal tears

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Complications of Instrumental Delivery

Forceps

  • Mother: lacerations to the cervix & vagina
  • Fetus: scalp lacerations and trauma to the head

Vacuum

  • Mother: lacerations/haematomas
  • Fetus:
    • Cephalohaematoma
    • Subgaleal haematoma (life-threatening)
    • Intracranial haemorrhage (life-threatening)

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Birth trauma

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Caput succedaneum:

  • Benign swelling
  • Crosses suture lines

Cephalhaematoma:

  • Benign
  • Doesn’t cross suture lines

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Subgaleal haematoma

  • Rupture of emissary veins (which connect dural sinuses & scalp veins)
  • May cross suture lines
  • Risk of substantial haemorrhage and haemorrhage shock

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Chignon (‘artificial caput succedaneum’)

  • Caused by collection of interstitial fluid and small haemorrhages, due to application of vacuum cup
  • May cross suture lines
  • Resolves within ~18hrs of birth
  • No long-term issues

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Intrapartum fetal monitoring

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Impact of labour on the fetus

  • With each contraction, placental blood flow and oxygen transfer are temporarily interrupted
    • Healthy fetuses can deal with this, but if already compromised before labour, may cause further problems
  • Insufficient oxygen delivery 🡪 switch to anaerobic metabolism 🡪 lactic acid & H+ ions 🡪 can lead to metabolic acidosis
  • Possible effects include neurological injury, fetal death

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Fetal assessment in labour

  • Colour of liquor (?fresh meconium ?heavy bleeding)
  • Fetal movement evaluation
  • Continuous external monitoring (CTG)
  • Doppler ultrasound
  • Fetal blood sampling

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When to initiate continuous CTG

  • When risk factors for fetal compromise identified
  • Use of IV syntocinon
  • Commence before epidural to establish baseline (and continue)

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Baseline

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Baseline: Mean FHR when stable

Determined over 5-10 minutes.

Normal: 110-160 bpm

  • Lower in the preterm fetus

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Baseline variability

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Baseline variability: minor FHR fluctuations.

Measure as bpm difference between highest peak & lowest trough in 1 minute

Normal/moderate: 5-25 bpm

Reduced/minimal: 3 – 5 bpm

Absent: <3 bpm

Increased/marked: >25 bpm

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Accelerations & Decelerations

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Causes of altered fetal heart rate: VEAL CHOP

Variable decelerations

Cord compression / prolapse

Early decelerations

Head compression

Accelerations

OK ☺

Late decelerations

Placental insufficiency

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Accelerations

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Accelerations: Transient increase in FHR of 15 bpm above baseline for ≥15 seconds (less than 10 mins)

Presence of 2+ accelerations in 20 minutes indicates a reactive FHR.

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Decelerations

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Decelerations: transient decrease in FHR of ≥15 bpm below baseline for ≥15 seconds.

Decelerations are interpreted in relation to contractions.

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Early decelerations

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Early decelerations:

  • Uniform, repetitive, slow-onset
  • Occur early in contraction and return to baseline by end of contraction.

Aetiology: Contraction compresses the fetal head, causing a vagal response.

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Variable decelerations

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Variable decelerations:

  • Repetitive or intermittent
  • Rapid onset and recovery
  • Variable relationship with contractions, although most commonly simultaneous with contractions

Aetiology: compression of cord / cord prolapse

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Late decelerations

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Late decelerations:

  • uniform, repetitive, usually slow-onset, at the middle to end of contraction
  • Lowest point >20 seconds after peak and ending after contraction

Aetiology: placental insufficiency, causing hypoxia & acidosis.

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Prolonged decelerations

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Prolonged deceleration: decrease of ≥15 bpm for longer than 90 seconds but less than 5 mins.

Aetiology:

  • Same as for variable & late decelerations, but lasting longer and more severe.

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CTG Features

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Features of reassuring CTG:

  • Baseline of 110 - 160bpm
  • Baseline variability of 5-25 bpm
  • Absence of decelerations

In labour, we are less worried about:

  • Absence of accelerations
  • Simple variable decelerations
  • Early decelerations

When to worry:

  • Fetal tachycardia or bradycardia
  • Loss of baseline variability
  • Recurrent variable decelerations or late decelerations

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Responding to an abnormal CTG

  • Reposition – left lateral position
  • IV fluids if hypotensive
  • Manual elevation of fetal head
  • Consider VE to exclude cord prolapse
  • Cease oxytocin, remove prostaglandin
  • Delay active pushing during 2nd stage of labour
  • Consider tocolytics
  • Emergency Caesarean if initial steps unsuccessful, or if significant fetal acidosis / compromise suspected.

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Caesarean Section

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Types of Caesarean Section

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This slide is just for understanding!

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Indications for Caesarean Section

  • Elective
    • Traditionally where labour & delivery may be detrimental to mother & fetus
    • Increasingly more women requesting for other reasons / personal preference
  • Emergency, most common indications are:
    • Failure to progress through first stage of labour
    • Fetal distress
    • Abnormal lie or malpresentation at start of labour

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Complications

  • Maternal
    • Bleeding
    • Thromboembolism
    • Injury to surrounding organs (bowel, bladder)
    • Infection
    • Complications for future pregnancies

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Impact of Caesarean on future pregnancies

  • Greater risk of uterine dehiscence and rupture
    • So future pregnancies recommended to be Caesareans as well, in many cases
    • Women who have had 2* or more Caesareans should have Caesareans for all future pregnancies
      • *3 according to PROMPT guidelines
  • Increased risk of placental adherence in future pregnancies, requiring a hysterectomy

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Vaginal Birth After Caesarean �(VBAC)

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Overview of VBAC

  • Avoiding major surgery & multiple Caesareans in future pregnancies
  • Able to mobilise & leave hospital sooner
  • Able to have a vaginal birth if patient wishes

  • 20-30% of women attempting VBAC will need Caesarean
  • Attempting VBAC carries an additional risk of uterine scar rupture and other complications

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Complications of VBAC

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Complication

Risk / 1,000 attempted VBACs

Uterine rupture

5-7

Perinatal death

0.4-0.7

Maternal death

0.02

Major maternal morbidity

- Hysterectomy

0.5-2

- Genitourinary injury

0.8

- Blood transfusion

1.8

Major perinatal morbidity

- Fetal acidosis (cord pH <7)

1.5

- Hypoxic-ischaemic encephalopathy (HIE)

0.4

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Complications of VBAC

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Complication

Risk / 1,000 attempted VBACs

Uterine rupture

5-7

Perinatal death

0.4-0.7

Maternal death

0.02

Major maternal morbidity

- Hysterectomy

0.5-2

- Genitourinary injury

0.8

- Blood transfusion

1.8

Major perinatal morbidity

- Fetal acidosis (cord pH <7)

1.5

- Hypoxic-ischaemic encephalopathy (HIE)

0.4

Uterine Rupture

Pathophysiology: uterus becomes distended and subsequently

ruptures.

Presentation:

- Maternal signs: severe abdo pain, PV bleeding, contractions stop

- Fetal signs: concerning CTG / decreased FHR, loss of station

- Maternal & fetal compromise

Many other complications of VBAC are due to uterine rupture.

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Complications of VBAC

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Complication

Risk / 1,000 attempted VBACs

Uterine rupture

5-7

Perinatal death

0.4-0.7

Maternal death

0.02

Major maternal morbidity

- Hysterectomy

0.5-2

- Genitourinary injury

0.8

- Blood transfusion

1.8

Major perinatal morbidity

- Fetal acidosis (cord pH <7)

1.5

- Hypoxic-ischaemic encephalopathy (HIE)

0.4

Hypoxic-ischaemic encephalopathy

- Neurological changes due to poor oxygen delivery perfusing

the fetal brain

- Manifests during first few days after birth

- Presentation: organ dysfunction (heart, lungs, kidneys, liver,

blood), seizures, lethargy, unresponsive.

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To VBAC or not to VBAC?

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To VBAC

Not to VBAC: ERCS

Pros

- Up to 80% success rate

- Quicker, easier recovery

- Avoid risk of uterine rupture and other intrapartum risks

Cons

- Increased risk of uterine rupture and associated complications

- Longer recovery

- Anaesthetic risks

- VTE risk

- Bleeding, infection risk

- Risk of injury to surrounding organs

Contraindications for VBAC

3+ previous C/S, need for IOL, previous labour suggesting cephalopelvic disproportion, previous classical Caesarean scar, previous uterine rupture, malpresentation (e.g. breech)

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Advice to improve VBAC Outcomes

  • Inter-pregnancy interval of at least 12 months
    • Better outcomes with further increased interval (see Monash PROMPT ‘Birth after a previous caesarean section’ guideline)
  • Weight reduction for overweight & obese patients
  • Manage delivery in obstetric unit with trained staff and continuous monitoring of maternal & fetal wellbeing

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Management of VBAC

  • CTG monitoring & regular VEs throughout labour
  • IV cannula
  • FBE, Group & Hold
  • Be careful with oxytocin (Consultant approval at Monash)
  • Do not use prostaglandins
  • Monitor for signs of uterine rupture and move to Caesarean if necessary

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Sources & Acknowledgements

  • O&G – An Evidence-Based Guide
  • Obstetrics by Ten Teachers 19E
  • PROMPT Guidelines
  • Monash Dept. Obstetrics & Gynaecology
  • RANZCOG Guidelines
  • AMBOSS
  • WHISM Online Resources

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THANKS!

Any questions?

Please feel free to email me ☺

skdew4@student.monash.edu

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