Labour & Delivery�Suwandi Dewapura�skdew4@student.monash.edu
Today’s Lecture
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Not covered here:
Stages of labour
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Stage 1:
0 🡪 10cm dilation
Active
Latent
A quick word about Rupture of Membranes (ROM):
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Stage 1:
0 🡪 10cm dilation
Management of normal Stage 1:
Active
Latent
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Active
Latent
Stage 1:
0 🡪 10cm dilation
Stage 2:
Fully dilated to birth of infant
During this stage:
Duration:
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Active
Passive
Stage 2:
Fully dilated to birth of infant
Presenting part descends through the pelvis (no urge to push)
Maternal effort to drive presenting part through birth canal
During this stage:
Duration:
Active
Latent
Stage 1:
0 🡪 10cm dilation
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Active
Passive
Stage 2:
Fully dilated to birth of infant
Management of normal Stage 2:
(indications include shoulder dystocia, instrumental delivery or vaginal breech)
Active
Latent
Stage 1:
0 🡪 10cm dilation
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Stage 3
Active
Passive
Stage 2:
Fully dilated to birth of infant
Stage 3:
Birth to expulsion of placenta
Active
Latent
Stage 1:
0 🡪 10cm dilation
During this stage:
Duration:
Management of normal Stage 3:
10 units oxytocin IM
Controlled cord traction (placenta separate on its own)
Management of prolonged labour
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Stage | Management |
Prolonged Stage 1 – Latent Phase | Conservative/expectant mx. Can consider ARM, cervical ripening or oxytocin. |
Prolonged Stage 1 – Active Phase | Augment labour with oxytocin if weak contractions; analgesia if strong contractions. +/- ARM, cervical ripening Caesarean if above management ineffective. |
Prolonged Stage 2 | Oxytocin if weak contractions or <1cm progress after 60-90 mins pushing. Trial of instrumental delivery if fetal head engaged & sufficient contractions. Caesarean if head not engaged / insufficient contractions. |
Prolonged Stage 3 | Uterotonic agent, controlled cord traction, empty bladder. IV access, FBE, Group & Hold. Manual removal of placenta (MROP). Hysterectomy if failure of MROP. |
Management of prolonged labour
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Stage | Management |
Prolonged Stage 1 – Latent Phase | Conservative/expectant mx. Can consider ARM, cervical ripening or oxytocin. |
Prolonged Stage 1 – Active Phase | Augment labour with oxytocin if weak contractions; analgesia if strong contractions. +/- ARM, cervical ripening Caesarean if above management ineffective. |
Prolonged Stage 2 | Oxytocin if weak contractions or <1cm progress after 60-90 mins pushing. Trial of instrumental delivery if fetal head engaged & sufficient contractions. Caesarean if head not engaged / insufficient contractions. |
Prolonged Stage 3 | Uterotonic agent, controlled cord traction, empty bladder. IV access, FBE, Group & Hold. Manual removal of placenta (MROP). Hysterectomy if failure of MROP. |
Management of prolonged labour
12
Stage | Management |
Prolonged Stage 1 – Latent Phase | Conservative/expectant mx. Can consider ARM, cervical ripening or oxytocin. |
Prolonged Stage 1 – Active Phase | Augment labour with oxytocin if weak contractions; analgesia if strong contractions. +/- ARM, cervical ripening Caesarean if above management ineffective. |
Prolonged Stage 2 | Oxytocin if weak contractions or <1cm progress after 60-90 mins pushing. Trial of instrumental delivery if fetal head engaged & sufficient contractions. Caesarean if head not engaged / insufficient contractions. |
Prolonged Stage 3 | Uterotonic agent, controlled cord traction, empty bladder. IV access, FBE, Group & Hold. Manual removal of placenta (MROP). Hysterectomy if failure of MROP. |
Management of prolonged labour
13
Stage | Management |
Prolonged Stage 1 – Latent Phase | Conservative/expectant mx. Can consider ARM, cervical ripening or oxytocin. |
Prolonged Stage 1 – Active Phase | Augment labour with oxytocin if weak contractions; analgesia if strong contractions. +/- ARM, cervical ripening Caesarean if above management ineffective. |
Prolonged Stage 2 | Oxytocin if weak contractions or <1cm progress after 60-90 mins pushing. Trial of instrumental delivery if fetal head engaged & sufficient contractions. Caesarean if head not engaged / insufficient contractions. |
Prolonged Stage 3 | Uterotonic agent, controlled cord traction, empty bladder. IV access, FBE, Group & Hold. Manual removal of placenta (MROP). Hysterectomy if failure of MROP. |
Induction of labour
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Induction of Labour (IOL)
Common indications
Contraindications
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Bishop’s Score
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If Bishop’s ≥5, cervix favourable for vaginal delivery.
Options for IOL
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Complications of IOL with Syntocinon
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Instrumental delivery
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Indications for Instrumental Delivery
Prerequisites for instrumental delivery
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Instrumental delivery
Vacuum
Forceps
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Instrumental delivery
Vacuum
Forceps
Both require:
Both can be used for management of:
Both contraindicated if:
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Instrumental delivery
Vacuum
Forceps
Complications of Instrumental Delivery
Forceps
Vacuum
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Birth trauma
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Caput succedaneum:
Cephalhaematoma:
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Subgaleal haematoma
Chignon (‘artificial caput succedaneum’)
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Intrapartum fetal monitoring
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Impact of labour on the fetus
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Fetal assessment in labour
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When to initiate continuous CTG
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Baseline
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Baseline: Mean FHR when stable
Determined over 5-10 minutes.
Normal: 110-160 bpm
Baseline variability
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Baseline variability: minor FHR fluctuations.
Measure as bpm difference between highest peak & lowest trough in 1 minute
Normal/moderate: 5-25 bpm
Reduced/minimal: 3 – 5 bpm
Absent: <3 bpm
Increased/marked: >25 bpm
Accelerations & Decelerations
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Causes of altered fetal heart rate: VEAL CHOP | |
Variable decelerations | Cord compression / prolapse |
Early decelerations | Head compression |
Accelerations | OK ☺ |
Late decelerations | Placental insufficiency |
Accelerations
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Accelerations: Transient increase in FHR of ≥15 bpm above baseline for ≥15 seconds (less than 10 mins)
Presence of 2+ accelerations in 20 minutes indicates a reactive FHR.
Decelerations
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Decelerations: transient decrease in FHR of ≥15 bpm below baseline for ≥15 seconds.
Decelerations are interpreted in relation to contractions.
Early decelerations
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Early decelerations:
Aetiology: Contraction compresses the fetal head, causing a vagal response.
Variable decelerations
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Variable decelerations:
Aetiology: compression of cord / cord prolapse
Late decelerations
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Late decelerations:
Aetiology: placental insufficiency, causing hypoxia & acidosis.
Prolonged decelerations
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Prolonged deceleration: decrease of ≥15 bpm for longer than 90 seconds but less than 5 mins.
Aetiology:
CTG Features
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Features of reassuring CTG:
In labour, we are less worried about:
When to worry:
Responding to an abnormal CTG
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Caesarean Section
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Types of Caesarean Section
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This slide is just for understanding!
Indications for Caesarean Section
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Complications
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Impact of Caesarean on future pregnancies
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Vaginal Birth After Caesarean �(VBAC)
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Overview of VBAC
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Complications of VBAC
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Complication | Risk / 1,000 attempted VBACs |
Uterine rupture | 5-7 |
Perinatal death | 0.4-0.7 |
Maternal death | 0.02 |
Major maternal morbidity | |
- Hysterectomy | 0.5-2 |
- Genitourinary injury | 0.8 |
- Blood transfusion | 1.8 |
Major perinatal morbidity | |
- Fetal acidosis (cord pH <7) | 1.5 |
- Hypoxic-ischaemic encephalopathy (HIE) | 0.4 |
Complications of VBAC
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Complication | Risk / 1,000 attempted VBACs |
Uterine rupture | 5-7 |
Perinatal death | 0.4-0.7 |
Maternal death | 0.02 |
Major maternal morbidity | |
- Hysterectomy | 0.5-2 |
- Genitourinary injury | 0.8 |
- Blood transfusion | 1.8 |
Major perinatal morbidity | |
- Fetal acidosis (cord pH <7) | 1.5 |
- Hypoxic-ischaemic encephalopathy (HIE) | 0.4 |
Uterine Rupture
Pathophysiology: uterus becomes distended and subsequently
ruptures.
Presentation:
- Maternal signs: severe abdo pain, PV bleeding, contractions stop
- Fetal signs: concerning CTG / decreased FHR, loss of station
- Maternal & fetal compromise
Many other complications of VBAC are due to uterine rupture.
Complications of VBAC
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Complication | Risk / 1,000 attempted VBACs |
Uterine rupture | 5-7 |
Perinatal death | 0.4-0.7 |
Maternal death | 0.02 |
Major maternal morbidity | |
- Hysterectomy | 0.5-2 |
- Genitourinary injury | 0.8 |
- Blood transfusion | 1.8 |
Major perinatal morbidity | |
- Fetal acidosis (cord pH <7) | 1.5 |
- Hypoxic-ischaemic encephalopathy (HIE) | 0.4 |
Hypoxic-ischaemic encephalopathy
- Neurological changes due to poor oxygen delivery perfusing
the fetal brain
- Manifests during first few days after birth
- Presentation: organ dysfunction (heart, lungs, kidneys, liver,
blood), seizures, lethargy, unresponsive.
To VBAC or not to VBAC?
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| To VBAC | Not to VBAC: ERCS |
Pros | - Up to 80% success rate - Quicker, easier recovery | - Avoid risk of uterine rupture and other intrapartum risks |
Cons | - Increased risk of uterine rupture and associated complications | - Longer recovery - Anaesthetic risks - VTE risk - Bleeding, infection risk - Risk of injury to surrounding organs |
Contraindications for VBAC
3+ previous C/S, need for IOL, previous labour suggesting cephalopelvic disproportion, previous classical Caesarean scar, previous uterine rupture, malpresentation (e.g. breech)
Advice to improve VBAC Outcomes
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Management of VBAC
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Sources & Acknowledgements
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THANKS!
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