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Introduction to Critical Care Nursing in Cardiac Surgery

Presented by Kim Duong-Coburn

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Learning Objectives

  • Summarize indications for cardiac surgery

  • Analyze principles of cardiopulmonary bypass

  • Apply preload, afterload, contractility concepts to achieve optimal cardiac output/index

  • Identify interventions to achieve hemodynamic stabilities using nursing critical thinking processes,

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Looking back…

  • 1892 Rehn in Frankfurt, Germany successfully repaired a L V stab wound with 3 sutures.

  • 1925 Souttar performed digital exam mitral valve on a beating heart via L chest wall incision on a 15 y/o who had severe mitral stenosis.

  • 1935 Beck rerouted the IMA to the surface of the heart to provide a collateral vascular bed on a 48 y/o coal miner suffering from debilitating cardiac pain.

  • 1949 Bailey pioneered valve commisurotomy to treat mitral stenosis.

  • 1954 John Gibbon from Philadelphia used “mechanical heart lung machine” on 18 y/o Cecilia BaVolek to repair her atrial septal defect.

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Cardiopulmonary Bypass

    • Principles of Cardiopulmonary Bypass:

      • Hemodilution
      • Hypothermia
      • Anticoagulation

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A=venous reservoir & blood filter

B=membrane oxygenator

C= heat exchanger coil

Cardiopulmonary Bypass Components

D= CPB console

E=cardioplegia reservoir

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Cannulation

Venous blood is drained into the oxygenator through 1-2 cannulas from R atrium ( or vena cava)

Arterial blood is returned via a single cannula in the ascending aorta

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Indications for Cardiac Surgery

  • CABG (coronary artery bypass grafting)
  • Valve repair / replacement
  • Thoracic aneurysm repair
  • Surgical management of arrhythmia
  • TMR (transmyocardial laser revascularization)
  • Ventricular reconstruction
  • Removal of myxoma
  • Surgical correction for congenital heart diseases
  • Insertion of ventricular assist device
  • Cardiac transplantation

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CABG

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CABG

  • Successful grafts typically last 8-15 years.
  • Younger patients with no complicating disease_better prognosis.
  • Cognitive decline postop_multiple factors (CPB, hypoxia, high/low body T, hyper/hypotension, arrhythmia)
  • Long term lifestyle modification, control BP, cholesterol, DB.

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CABG

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Valvular Heart Disease

  • Stenosis vs. regurgitation

  • Repair vs. replacement

  • Biological vs. mechanical valve

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Heart Valves

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Mitral Stenosis

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Postop Care

  • “all postop cardiovascular patients are considered unstable during the first 6 hours unless proven otherwise”

(Cabral et al. Manual of Postoperative Management in Adult Cardiac Surgery)

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Postop Care

5 “headaches

  • Hypothermia
  • Hypovolemia
  • Hypotension
  • Hypertension
  • Hemorrhage

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Hypothermia

  • Rewarming: T raised to 36-37 C before end of CPB
  • After drop: 45 min postop in CCU
  • Overshoot: 8-12 hrs postop, up to 38.3 C
  • Shivering
  • Rewarm acidosis: “wash out” of lactic acid
  • Rewarming shock

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Hypovolemia

  • Volume replacement: crystalloid,colloids,blood products

  • Frequent causes= hemmorhage,diuresis, third spacing

  • Compensatory responses to hypovolemia= increase SVR, increase HR, increase myocardial contractility, shifting fluid into intravascular compartment

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Hypotension

  • Volume replacement: albumin, LR, blood products
  • Inotropic supports
  • Prevention of warming shock
  • Control postop hemorrhage
  • Treat warming acidosis
  • Improve cardiac measures

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Hypertension

  • Paroxymal postop hypertension: immediate postop to 4-6 hrs after
  • Parameters: SBP>130-160, DBP>90-100, MAP>90-110 mmHg
  • Intervention: pharmacology support, correct hypoxia/hypercapnia, adequate volume
  • Nipride metabolized into cyanogen which is converted to thiocyanate in liver, excreted by kidney

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Hemmorhage

  • Sequalae of CPB on coagulation
  • Systemic Inflammatory Response Syndrome
  • Bleeding 3ml/kg/hr
  • Keep SBP 90-100, MAP 60-65 unless hx CVA, carotid disease
  • Peep at 10 cm if CO,CI adequate
  • Auto transfusion
  • Blood products and pharmacolgical support

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Other consideration

  • Goal = maintain adequate tissue perfusion to prevent end organ failure
  • Keep adequate CO,CI (pace,preload,afterload,contractility)
  • Maintain adequate elctrolyte
  • Prevent arrhythmia
  • Tight control hyperglycemia
  • Early extubation as tolerated

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Cardiac output

  • CO = a measure of the amount of blood that is ejected by the heart each minute.
  • SV = represents the amount of blood that is ejected from the LV with one contraction.
  • CO = SV X HR

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Cardiac output

  • SV is influenced by amount of blood in the ventricle and the force of contraction by the ventricle.

  • SV also affected if aortic valve restricted the outflow of the LV

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Ejection fraction (EF)

  • EF = is the percentage of the volume of the LV that is ejected with each contraction.

  • Normal EF = 65-70%

  • EF reflects the efficiency of LV to pump blood forward into the systemic circulation

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Hemodynamic

Parameters

Formula

Normal Ranges

Cardiac Output

CO

CI

HR X SV

CO/BSA

4-8 L/min

2.5-4 L/min./m2

Preload Indicators

CVP

Wedge

LA

2-6 mmHg

8-12 mmHg

4-12 mmHg

Afterload Indicators

PVR

SVR

(PAM-PAWPX80)/CO

(MAP-CVPX80)/CO

37-250 dynes/sec/cm5

800-1200 dynes/sec/cm5

Contractility Indicators

SI (Stroke Volume Index)

RVSWI

LVSWI

SV/BSA

(PAM-CVP)SV x 0.0136/BSA

(MAP-PAD)SVx0.0136/BSA

33-47 cc/m2/beat

7-12 g/m2/beat

38-85g/m2/beat

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Key Concepts in Measuring CO

  • Accuracy depend on functioning tricuspid, no ventricular septal defect,and a stable rhythm

  • Correlate CI with a tissue oxygenation parameter (Svo2(60-70%),lactate(1-2 mEQ/L), pH(7.35-7.45),HCO3 (22-26 mEQ/L)

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Key Concepts in Measuring CO

  • LVEDV =100ml, SV =80ml, EF=80%(norm.=65-70%) LV dysfunction (dilatation): LVEDV=110,SV=70,EF=64%(70/110)

  • LVEDV=170,SV=70,EF=41%(70/170)

  • LVEDV and EF are early warning signs for LV dysfunction

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Preload

  • Affected most adversely during hypovolemia, vasodilatation by fluid shift, hemorrhage, inflammatory responses, and rewarming

  • Common preload reduction agents are diuretics and NTG(vasodilation by venous pooling lead to reduce preload, improve LV contractility and myocardial blood flow)

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Afterload

  • Reducing afterload lead to decrease LV work therefore improve LV contractility and reduce myocardial consumption

  • Afterload reducers are used initially only if high BP and/or high SVR are the cause of LV dysfunction

  • Afterload reducers are used after inotropic therapy and after preload reduction

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THANK YOU

Questions?