Electrical injury Canada
Electrical Injury in Canada – 1999-2009
Work Categories
Type of work: | (2006-2012) |
Electrician | 31% |
Journeyman/Lineman | 17% |
Technician: maintenance, light, HVAC | 14% |
Other: Building maintenance/supervisor, millwright, welder, boom crane operator, water operator in training, rough carpentry, crusher operator, electrical engineer | 45% |
EI Patient Clinical Presentation
Electrical injury
Low Voltage injury
NOTHING VISIBLE
| Low-Voltage (ntotal) | High-Voltage (ntotal) | Voltage Not Otherwise Specified (ntotal) | p-value (LV vs. HV) |
Surgical intervention (%)† | 54.1 (652) | 79.6 (608) | 66.1 (587) | 0.004* |
Fasciotomy/Escharotomy (%)† | 4.8 (395) | 27.0 (1019) | 15.7 (922) | 0.2 |
Amputation (%)† | 7.3 (640) | 30.2 (1898) | 27.2 (1501) | 0.02* |
Compartment Syndrome (%)† | - | 15.0 (120) | - | - |
Reconstructive Flap (%)† | - | 114.8 (684) | 12.5 (585) | - |
Average Length of Stay (days) | 10.9 (600) | 31.2 (1717) | 19.8 (1898) | 0.002* |
ECG changes (%) | - | 20.0 (315) | 14.6 (941) | - |
Myoglobinuria (%) | - | 38.9 (476) | 27.3 (362) | - |
Renal dysfunction (%) | 0.0 (108) | 13.9 (1098) | 4.2 (873) | 0.4 |
Infection (non-specific) (%) | - | 15.0 (832) | 25.5 (553) | - |
Mortality (%) | 2.6 (887) | 5.2 (1755) | 3.7 (1944) | 0.2 |
Mortality (n, %) | 23 | 92 | 71 |
|
Causes of Mortality* |
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|
|
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TBSA > 50% (n, %) | - | 12 (48%) | 10 (20%) |
|
Multi-organ failure/ � Septicemia (n, %) | - | 11 (44%) | 7 (14%) |
|
Pneumonia (n, %) | - | 0 (0%) | 8 (15%) |
|
ARDS (n, %) | - | 1 (4%) | 0 (0%) |
|
Renal Failure/ATN (n, %) | - | 0 (0%) | 14 (27%) |
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Myocardial infarction/ � Cardiopulmonary arrest � (n, %) | - | 1 (4%) | 3 (6%) |
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Ventricular fibrillation � (n , %) | - | 0 (0%) | 9 (18%) |
|
Hepatic failure (n, %) | - | 1 (4%) | 0 (0%) |
|
Shih, Shahrokhi, Jeschke. JBCR 2017
Results – Mortality
Mortality
| All Patientsa | HVI | LVI | P
|
No. of patients | 207 | 76 | 122 |
|
LOS, days, median (IQR) | 9 (3-18) | 14 (4-24) | 8 (3-15) | <0.001 |
LOS/TBSA, days/%, median (IQR) | 2 (1-4) | 3 (1-8) | 2 (1-3) | <0.001 |
TBSA, %, median (IQR) | 4 (1-10) | 3 (1-15) | 5 (2-9) | .44 |
No. of ORs, median (IQR) | 1 (0-2) | 2 (0-3) | 0 (0-1) | <.001 |
Complications, no. (%) |
|
|
|
|
Rhabdomyolysis | 9 (4) | 9 (12) | 0 (0) | <.001 |
Compartment syndrome | 17 (8) | 12 (16) | 5 (4) | .007 |
Infection | 28 (14) | 15 (20) | 11 (9) | .05 |
Sepsis | 11 (5) | 8 (11) | 3 (3) | .02 |
Multiple organ failure | 1 (1) | 1 (1) | 0 (0) | .38 |
Amputation | 26 (13) | 21 (28) | 3 (2) | <.001 |
Multiple amputations | 13 (6) | 10 (13) | 2 (2) | .001 |
Requiring rehabilitation, no. (%) | 106 (51) | 49 (65) | 54 (44) | .008 |
Discharged to inpatient rehabilitation, no. (%)b | 34 (32) | 22 (45) | 10 (19) | .005 |
Discharged to outpatient rehabilitation, no. (%)b | 72 (68) | 27 (55) | 44 (81) | .005 |
Mortality, no. (%) | 4 (2) | 3 (4) | 1 (1) | .16 |
Abbreviations: HVI, high-voltage injury; LVI, low-voltage injury; LOS, length of stay; TBSA, total body surface area. a Includes patients whose voltage was not otherwise specified (n=9). b Percentages are calculated based on the total number of patients requiring any form of rehabilitation (All patients, n=106; HVI, n=49; LVI, n=54). | ||||
Clinical outcomes during the acute phase of EI management.
| All Patientsa | HVI | LVI | P
|
Acute Cohort |
|
|
|
|
No. of patients | 59 | 26 | 31 |
|
Neuropsychological sequelae, no. (%) | 14 (24)
| 6 (23)
| 7 (23)
| >.99
|
Long-Term Cohort |
|
|
|
|
No. of patients | 122 | 51 | 69 |
|
Days to first follow- up, median (IQR)b | 201 (68-766) | 504 (179-1236) | 224 (41-333)
| <.001 |
Days to last follow-up, median (IQR)b | 980 (391-1409) | 1099 (511-1651) | 773 (315-1218) | .02
|
Neuropsychological sequelae, no. (%) |
|
|
|
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<5 yrs. post-injury | 99 (81) | 42 (82) | 56 (81) | >.99 |
>5 yrs. post-injuryc | 20 (27) | 13 (35) | 7 (20) | .19 |
Psychological/ Psychiatric treatment, no. (%) | 78 (64)
| 31 (61)
| 47 (68)
| .44
|
Medication, no. (%) | 78 (64) | 30 (59) | 47 (68) | .34 |
Abbreviations: HVI, high-voltage injury; LVI, low-voltage injury. Analysis excludes patients with documented pre-existing psychiatric conditions. a Includes patients whose voltage was not otherwise specified (acute cohort, n=2; long-term cohort, n=2). b Calculated from the date of injury. c Percentages are calculated based on the total number of patients that were available for follow-up at >5 years post-injury (All patients, n= 74; HVI, n=37; LVI, n=35).
| ||||
Neuropsychological sequelae and management.
Neuropsychological symptoms of EI during the acute phase
Neuropsychological symptoms of EI during the long-term phase
| All Patientsa | HVI | LVI | P
|
No. of patients | 90 | 39 | 49 |
|
Return to pre-injury occupation, no. (%) | 55 (61) | 23 (59) | 30 (61) | >.99
|
Modified Duties, no. (%)c | 33 (60) | 15 (65) | 17 (57) | .58 |
Modified Schedule, no. (%)c | 30 (55) | 11 (48) | 17 (57) | .59 |
Labour Market Re-Entry, no. (%) | 17 (19) | 9 (23) | 8 (16) | .80 |
Time to RTW, days, median (IQR)b | 166 (82-414) | 207 (102-548) | 124 (57-348) | .12 |
Unable to RTW, no. (%) | 17 (19) | 6 (15) | 11 (22) | .43 |
Abbreviations: HVI, high-voltage injury; LVI, low-voltage injury; RTW, return-to-work. a Includes patients whose voltage was not otherwise specified (n=2). b Calculated from the date of injury. c Percentages are calculated based on the total number of patients that returned to their pre-injury occupation (All patients, n= 55; HVI, n=23; LVI, n=30).
| ||||
Return-to-work characteristics of occupational EIs long-term cohort.
Symptomatology - Diffuse Electrical Injury
Long Term Sequelae of Electrical Injuries from Literature (cont’d)
Symptomatology - Diffuse Electrical Injury
80% of Specialty Consultations were negative
72% of Diagnostic Tests were negative
Arch Phys Med Rehabil 2012;93:623-628
CAUSE ?
Channelopathy
The electrical current makes tiny holes (called nano pores) in the walls of cells. These holes cause the cells to leak like a sinking ship. The cells take on water, lose their contents and try to keep up but eventually they cannot keep up and cells die.
Initial damage is isolated to the path that receives the highest current. As time progresses, surrounding cells previously dependent on damaged cells no longer receive nutrition, pulses, oxygen from microstructures damaged by electricity, slowly they starve and begin to die off, consequently causing additional neurodegeneration.
An electric shock by a single 4 ms and 500 mV pulse may decreased 20% of the Na+ channels and 30% of the K+ channels.
Immediate & delayed effects
What can we do now?
Awareness of Society and Medical Field
Identify channels
Lifestyle changes
Dietary modifications
Awareness of Triggers
Building Bridges